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EKG PATOLOGICA
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SCHEMA RAPIDA DEINTERPRETARE A EKG
1. FRECVENTA
2. RITMUL3. AXA4. HIPERTROFIE
5. INFARCT
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1.FRECVENTA
MODIFICARI DE FRECVENTA
I. BRADICARDIEfrecvente lente < 60 batai/minut
* ritm jonctional
II. TAHICARDIE
frecvente rapide > 100 batai/minut( 100 150 / min )
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I.BRADICARDIE SINUSALA
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Bradicardie sinusala
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RITM JONCTIONAL
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II. TAHICARDIE SINUSALA
- frecventa rapidaintre 100 si 150/minut- unde P identice
inainte de fiecare
complex QRS- ritm regulat
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Tahicardie sinusala
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2. RITM
TULBURARI DE RITM
I. RITM NEREGULATII. EXTRASISTOLE
III. RITMURI RAPIDE
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I . RITM NEREGULAT
1. ARITMIA SINUSALA
- unda de stimulare ia nastere in NSAdar impulsurile sunt eliberate la intervalevariabile
- unde P identice- distante R-R inegale
- se intalneste in mod normal la copii- la adulti, adesea se datoreaza bolii
nodului sinusal (sick sinus syndrome)
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ARITMIE SINUSALA
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ARITMIA SINUSALA
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2. RITM VAGABOND( wandering pacemaker )
- acest ritm apareca urmare a schimbariipozitiei pacemaker-ului
- unde P diferiteca forma in functie desediul pacemaker-ului
- distante R-R
inegale
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WANDERING PACEMAKER
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RITM VAGABOND
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3. FIBRILATIA ATRIALA
- se datoreazadescarcarii a numeroasefocare atriale ectopice sinumai din intamplare unul
dintre impulsuri ajunge laNAV- nu exista unde P- exista mici unde de
fibrilatie ( f )- distante R-R
inegale
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FIBRILATIA ATRIALA
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FIBRILATIA ATRIALA
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II. EXTRASISTOLE
Sunt batai care apar maiprecoce decat normal.
Se datoresc unor descarcaripremature a diferitelor focareectopice atriale sau ventriculare.
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1. EXTRASISTOLA ATRIALA
-un focar atrial ectopic
genereaza unde P anormale,precoce urmate de cate un
QRS normal. Impulsul nu ia
nastere in NSA deci nu seaseamana cu celelalte undeP. In schimb, NAV capteazasi transmite impulsul normal.
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EXTRASISTOLA ATRIALA
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2. EXTRASISTOLA NODALA
- un focar ectopic al noduluiatrio-ventricular genereaza unQRS precoce, neprecedat de unda Psau precedat de unda P negativa
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3. EXTRASISTOLA VENTRICULARA
EsV ia nastere intr-un focar ectopic al unuiventricul. Se caracterizeaza printr-un complex QRS largsi precoce urmat de o pauza compensatorie.
In contrast cu EsV, EsA sunt urmate de o pauza care nu este
compensatorie.
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EXTRASISTOLA VENTRICULARA
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EsV se cupleaza uneori cu una saumai multe batai normale si acest aspect seproduce regulat. Cand se cupleaza o EsV cuo bataie normala este vorba de bigeminism,cu doua batai trigeminism etc.
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Bigeminism
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Trigeminism
Un focar ventricular ectopic se poate
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Un focar ventricular ectopic se poatedescarca o singura data sau din contra saproduca o serie de impulsuri succesive care
realizeaza o salva de extrasistole.
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EsV multifocale se datoresc focarelorectopice ventriculare multiple. EsV multifocale au
morfologie diferita.
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III. RITMURI RAPIDE
1. TAHICARDIA PAROXISTICA
Inseamna o frecventa rapida( tahicardie ) cu debut brusc( paroxistica ).
Frecventa cardiaca in TPeste de 150 250/minut.
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TAHICARDIA PAROXISTICA ATRIALA
- se datoreaza unei descarcari rapide si brustea unui focar ectopic atrial.- este o succesiune normala de unde P si
complexe QRS cu o frecventa foarte rapida
- TPA poate sa se produca la o frecventa atat derapida incat undele P se amesteca cu undele Tceea ce da aspectul unei unde unice.
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TAHICARDIA PAROXISTICA NODALA
- se datoreaza unui focar ectopic situat in NAV- este o succesiune de complexe QRSneprecedate de unda P cu o frecventa foarterapida.
- TPA si TPN sunt denumite in ansamblutahicardie paroxistica supraventriculara
( TPSV )- diagnosticul TPA sau TPV nu este esential,
ele se trateaza la fel.
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TAHICARDIA PAROXISTICAVENTRICULARA
- este declansata de un focar ectopicventricular
- seamana cu o succesiune rapida de
EsV- frecventa rapida ( 150 250/minut )
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2. FLUTTER ATRIAL
- ia nastere intr-un focar ectopicial unic
- undele de flutter ( F ) survin inccesiune rapida si sunt identice
- sunt comparate cu dintii derastrau
- ritmul este in general regulat- frecventa undelor de flutter este
prinsa intre 250 350/minut- impulsul atrial va stimula periodic
V ceea ce inseamna ca va exista orie de unde F inaintea fiecarui complexS
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FLUTTER ATRIAL
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3. FLUTTER VENTRICULAR
- este produs de un focar ectopicventricular unic
- undele sinusoidale regulate se
succed cu o frecventa intre 200 300/minut
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4. FIBRILATIA ATRIALA
- se datoreaza numeroaselor focareectopice atriale care se descarca cu frecventediferite antrenand un ritm haotic , neregulat
- nu exista unde P ci unde f (de fibrilatie)- numai din intamplare un impuls atrial va
stimula nodul AV
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FIBRILATIA ATRIALA
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5. FIBRILATIE VENTRICULARA
-se datorestestimulilor ce iau nastere infocare ectopice ventricularemultiple antrenand contractii
haotice- activitatea electricaeste complet anarhica
- amplitudineadeflexiunilor diminua pemasura ce inima moare
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FIBRILATIE VENTRICULARA
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EXERCITII
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?
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FLUTTER ATRIAL
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?
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FLUTTER VENTRICULAR
?
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?
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1. TAHICARDIE SINUSALA
2. TAHICARDIE
SUPRAVENTRICULARA3. TAHICARDIE VENTRICULARA
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?
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EsA
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?
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EsN
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EsAEsNEsV - bigeminism
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?
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RITM SINUSAL NORMAL
?
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?
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ARITMIE SINUSALA
?
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?
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TAHICARDIE SINUSALATAHICARDIE VENTRICULARA
?
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?
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BRADICARDIE SINUSALA
?
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?
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WANDERING PACEMAKER
?
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?
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EXTRASISTOLE VENTRICULARE
?
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?
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FIBRILATIE ATRIALA
?
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?
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FIBRILATIE VENTRICULARA
TULBURARI DE CONDUCERE
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TULBURARI DE CONDUCERE
1. BLOC SINOATRIAL
2. BLOCURI ATRIOVENTRICULARE3. BLOCURI DE RAMURA
1 BLOC SINOATRIAL
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1. BLOC SINOATRIAL
- NSA este blocat temporar- pacemaker-ul normal al cordului se
blocheaza temporar cel putin in timpul unui cicludar apoi isi reia activitatea de stimulare.
- undele P inainte si dupa bloc sunt identice( toate iau nastere in NSA )
2 BLOCURI ATRIOVENTRICULARE
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2. BLOCURI ATRIOVENTRICULARE
BAV gradul I- impulsul intarzie la nivelul NAV mai mult
decat normal ( 0,1 sec )
- intervalul PQ/PR > 0,20 sec- de indata ce NAV este stimulat,depolarizarea ventriculara se produce normal
- secventa QRS T normala
BLOC AV GRADUL I
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BLOC AV GRADUL I
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BAV d l II
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BAV gradul II
Un bloc AV apare atunci cand suntnecesare mai mult de 2 impulsuri atrialepentru a declansa un raspuns ventricular.
Asta inseamna ca doua sau mai multeunde P preced un complex QRS
BAV gradul II Mobitz I
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BAV gradul II Mobitz I( perioada WencKebach )
- apare cand intervalul PR se alungesteprogresiv pana apare un P blocat
BAV gradul II Mobitz I
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g adu ob t
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BAV grad l II Mobit II
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BAV gradul II Mobitz II
- se constata absenta ocazionala aunui complex QRS dupa o unda P normala
- intervalele PR sunt normale
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BAV gradul III
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BAV gradul III
- apare atunci cand niciunul dintre impulsurileatriale nu stimuleaza NAV- ventriculele nestimulate pun in actiune un
pacemaker ectopic de la nivelul lor care va descarcaimpulsuri cu frecventa ~ 30/min
- in BAV grd III se constata ca frecventa atriala( undele P ) difera de cea ventriculara ( complexeleQRS ). Aceasta se numeste disociatie atrioventriculara.
- In BAV gradul III pulsul este atat de lent incatfluxul sangvin cerebral este diminuat si pacientul poateprezenta pierderea constientei
BAV COMPLET
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BAV COMPLET
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Bloc AV gradul I
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3 BLOCURI DE RAMURA
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3. BLOCURI DE RAMURA
- se datoreaza unui blocaj al impulsului inramurile dreapta si stanga a fasciculului Hiss
- ramura dreapta trimite stimulul dedepolarizare la ventriculul drept iar ramura stangala ventriculul stang. Acest lucru se realizeaza in
mod normal simultan.- un bloc al unei ramuri determina o
intarziere a transmiterii impulsului de partearespectiva. Deoarece ventriculele nu se mai
depolarizeaza simultan , QRS ul va avea aspectlargit > 0,12 sec. Depolarizarea fiecarui ventriculse realizeaza separat astfel ca pe EKG apar douaunde R care se noteaza R si R
Daca gasim un QRS largit peste 0,12 sec peEKG inseamna ca exista un bloc de ramura.
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EKG inseamna ca exista un bloc de ramura.Cautam aspectul R- R in precordiale.
- R- R in precordialele drepte (V1 si V2)- axa la dreapta BRD
- ST si T in sens opus QRS
- R- R in precordialele stangi ( V5 si V6 )
- axa la stanga BRS- ST si T in sens opus QRS
BLOC DE RAMURA DREAPTA
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BLOC DE RAMURA DREAPTA
BLOC DE RAMURA STANGA
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BLOC DE RAMURA STANGA
BLOC DE RAMURA DREAPTA
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BLOC DE RAMURA DREAPTA
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BLOC DE RAMURA STANGA
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BLOC DE RAMURA STANGA
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SDR DE PREEXCITATIE( WOLFF-PARKINSON-WHITE )
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( WOLFF PARKINSON WHITE )
- reprezinta o depolarizare prematura a
ventriculelor- pe EKG se inscrie un interval PR scurtat si unda
delta
- la indivizii cu sdr WPW exista un fascicul
accesoriu care elimina intarzierea obisnuita de 0,1 sec aimpulsului la nivelul NAV
- acesti indivizi pot dezvolta usor TPSV
SDR DE PREEXCITATIE( WOLFF PARKINSON WHITE )
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( WOLFF-PARKINSON-WHITE )
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EXERCITII
?
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?
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BLOC SINOATRIAL
?
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?
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BAV gradul II Mobitz I
BAV gradul II Mobitz II
?
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?
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BAV gradul I
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MOBITZ I
BSA
MOBITZ II
?
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?
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EXTRASISTOLA ATRIALA
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TAHICARDIE SINUSALA
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SDR DE PREEXCITATIE
?
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?
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EXTRASISTOLEVENTRICULARE
?
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?
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BAV gradul III ( complet )
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FIBRILATIE ATRIALA
?
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TAHICARDIE
SUPRAVENTRICULARA
?
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TAHICARDIE VENTRICULARA
?
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RITM JONCTIONAL
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BRS
?
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FLUTTER VENTRICULAR
?
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BLOC SINOATRIAL
?
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FIBRILATIE VENTRICULARA
?
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EXTRASISTOLA ATRIALA
?
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FLUTTER ATRIAL
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HIPERTROFIE
SIINFARCT
4. HIPERTROFIE
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Hipertrofia unei cavitati inseamna ocrestere a grosimii peretelui acelei cavitati.
HIPERTROFIE ATRIALA
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Pentru evidentierea hipertrofieiatriale se examineaza unda P. Daca
unda P este inalta > 2,5 mV saumasoara > 3 patrate mici ( > 0,12sec ) atunci exista hipertrofie atriala.
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HIPERTROFIE ATRIALASTANGA
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HAS
Aspect bifid alundei P cu primacocoasa mai joasa si adoua mai inalta. Bazaundei P depaseste 0,12sec. Se mai numesteP mitral sau P bifid sise intalneste in stenozamitrala
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HIPERTROFIA VENTRICULARADREAPTA
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HVD
- unda R mare in V1si V2
- unda R diminuaapoi progresiv spre V4- complex QRS larg( 0,12 sec )
- deviatie axialadreapta
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HAD si HVD
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HIPERTROFIA VENTRICULARASTANGA
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HVS- S in V1 + R in V5 35 mm
= indicele Sokolov - Lyon
- deviatie axiala stanga- complex QRS larg( 0,12 sec )
- unda T inversata
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5. INFARCT
Infarctul miocardic este
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Infarctul miocardic este
consecinta unei obstructii a uneiartere coronare.
Ventriculul stang care areperetele cel mai gros are nevoiede o irigatie sangvina
importanta si este primul caresufera de pe urma diminuariicirculatiei coronare.
Zona infarctata este din
punct de vedere electric moartasi nu poate conduce impulsurielectrice.
TRIADA CLASICA AINFARCTULUI
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ISCHEMIE
( T negativ )
LEZIUNE(ST supradenivelat)
NECROZA( Q patologic )
DIAGNOSTICUL POZITIV ALINFARCTULUI DE MIOCARD
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Diagnosticul pozitiv al infarctului de miocardse pune pe existenta celor trei criterii :
1 ) necroza : unda Q patologicalargime = 0,04 sec
adancime = 1/3 1/4R
2 ) leziune : ST supradenivelat, cu formaarcuata, concava spre linia izoelectrica a traseului
3 ) ischemie : T negativ, amplu
DIAGNOSTICUL TOPOGRAFIC ALINFARCTULUI MIOCARDIC
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I. ANTERIOR : Q patologic in V1, V2, V3, V4
I. LATERAL : unde Q patologice in DI , aVL
I. INFERIOR : Q patologic in DII, DIII, aVF
I. POSTERIOR : unda R mare in V1, V2subdenivelare ST in V1, V2
INFARCT ANTERIOR : Q patologic inV1, V2, V3
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INFARCT LATERAL : unde Q patologicein DI , aVL
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INFARCT INFERIOR : STsupradenivelatin DII, DIII, aVF
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INFARCT POSTERIOR:unda R mare inV1,V2subdenivelare ST in V1, V2
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DIAGNOSTICUL EVOLUTIV ALINFARCTULUI MIOCARDIC
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Se disting 4 stadii :Stadiul I ( precoce )
faza supraacuta
Stadiul II ( intermediar ) faza acutaStadiul III ( tardiv )
faza de rezolutie
Stadiul IV ( cicatricial ) faza cronica
Stadiul I( precoce , supraacut )
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- apare in primele 24 36de ore de la debut
- supradenivelare foarteampla de ST
- segmentul ST pleaca dinvarful undei R
- unda S nu mai existaeste contopita cu unda T carenici ea nu se mai distinge
( unda in dom )- apare Q patologic
Stadiul II( intermediar , acut )
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- apare in prima saptamana dela debut
- aspect EKG caracteristic :unda Q patologica,segmentul ST supradenivelatsi unda T negativa
- pe zi ce trece, segmentul STcoboara tot mai mult spre
linia izoelectrica iar unda Tdevine tot mai ampla
Stadiul III( tardiv, de rezolutie )
apare la 1 3 sapt de la
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177/242
- apare la 1 3 sapt de la
infarct- pe EKG coexista unda Q
patologica si unda T negativa,ampla
( T coronarian )- segmentul ST devineizoelectric
- dupa 3 saptamani, unda T
negativa incepe sa semicsoreze treptat
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Stadiul IV( cronic, cicatricial )
- persista indefinitdupa vindecarea clinicaa infarctului
- pe EKG apare doarunda Q patologica
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ANGINA PECTORALA
DGN electrocardiografic
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DGN electrocardiografic
- Modificari de tip ischemic: subdenivelare ST
- Unda T turtita, uneorinegativa
- In unele cazuri aspectulEKG de angina pectoralaapare numai dupa probade efort
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181/242
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182/242
EXERCITII
?
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183/242
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184/242
BLOC SINOATRIAL
?
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185/242
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186/242
BAV gradul II Mobitz I
BAV gradul II Mobitz II
?
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187/242
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188/242
BAV gradul I
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189/242
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190/242
MOBITZ I
BSA
MOBITZ II
?
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191/242
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192/242
EXTRASISTOLA ATRIALA
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193/242
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194/242
TAHICARDIE SINUSALA
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195/242
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196/242
SDR DE PREEXCITATIE
?
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197/242
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198/242
EXTRASISTOLE
VENTRICULARE
?
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199/242
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200/242
BAV gradul III ( complet )
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201/242
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202/242
FIBRILATIE ATRIALA
?
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203/242
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TAHICARDIE
SUPRAVENTRICULARA
?
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205/242
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206/242
TAHICARDIE VENTRICULARA
?
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207/242
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208/242
RITM JONCTIONAL
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209/242
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210/242
BRD
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211/242
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212/242
BRS
?
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213/242
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214/242
FLUTTER VENTRICULAR
?
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215/242
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216/242
BLOC SINOATRIAL
?
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217/242
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218/242
?
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219/242
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220/242
FLUTTER ATRIAL
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221/242
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222/242
INFARCT LATERAL
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223/242
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224/242
HAD si HVD
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225/242
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226/242
HVS
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227/242
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228/242
INFARCT INFERIOR
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229/242
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230/242
HAD
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231/242
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INFARCT ANTERIOR
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233/242
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ANGINA PECTORALA
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235/242
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HAS
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237/242
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238/242
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239/242
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240/242
Click to edit Master subtitle style
HEMIBLOC ANTERIOR
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241/242
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Click to edit Master subtitle style
HEMIBLOC POSTERIOR
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